I Lost my father to Dementia on June 9th, 2020.

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My family originally came from Wallachia in Europe
Dementia Prevention Systems, Inc.

My Father suffers from Dementia. I don’t know how much longer we will have him. He has been a guiding light in darkness his entire life. Having raised 7 kids and putting each through college, he also fed and clothed and housed several uncles and my now deceased grandma and grandpa. He served in the United States Military in the Army and was active in the Czech heritage society and helped build the Czech heritage museum in La Grange. I owe him so much. Dementia is cruel. It takes dignity and recognition away. It leaves fear and anguish. My father fights everyday. Every day my brothers monitor and attend to him 24hrs a day. Cleaning, baths, feeding, walking several times a day. Walking is critical for sufferers of Alzheimer’s and Dementia.  I wanted you to meet my father. He would have treated you as his own child should you have walked through the door with me. I Love him with all my heart. I hope I will make him proud.

My father died on June 8th, 2020. He was my best friend. I will miss him more than I can ever express..
devastated

Texas Czech Heritage and Cultural Center

Dementia is not well understood. We are just starting to understand that many factors and types of Dementia exist. Potential treatments will likewise be varied and customized to the individual. If you have Dementia in your family PLAN NOW!
Dementia is exceptionally cruel and treatments over time are expensive
Diagnosis of Dementia is increasing each year!

THERE IS NO CURE. SOUND FAMILIAR?

DEMENTIA IS ON THE RISE

The incidence of dementia increased by 117% between 1990 and 2016, according to the study findings. The results showed that globally, the number of prevalent dementia cases increased from 20.2 million in 1990 to 43.8 million in 2016 between 1990 and 2016

Every year, there are nearly 10 million new cases. … The total number of people with dementia is projected to reach 82 million in 2030 and 152 in 2050. Much of this increase is attributable to the rising numbers of people with dementia living in low- and middle-income countries.

A fire is spreading in our society and we have no way to put out this fire. Our time is running out. We must change society completely to deal with the problem of Dementia and Dementia related illnesses. Our society is becoming more poisonous and causing more genetic damage.
VPM News
in the news 2020

FierceBiotech EIP Pharma tees up phase 3 for ex-Vertex dementia drug

Grazia Video Shows Former Ballerina With Dementia Remembering Swan Lake

MedPage Today Infection-Alzheimer’s Link? Concussion Spit Test

UPDATE

Scientists at Washington University School of Medicine (WUSM) in St. Louis have spent some years investigating the links between circadian rhythm and Alzheimer’s, and have recently been making some real inroads. Following a 2018 study demonstrating how disrupted sleep can accelerate the buildup of toxic plaques associated with the disease, the team has now identified a protein implicated in the progression of the disease that appears highly regulated by the circadian rhythm, helping them join the dots and providing a potential new therapeutic target.

The brain protein in question is called YKL-40 and for years has served as a biomarker for Alzheimer’s, as high levels of it have been found in the cerebrospinal fluid of those suffering from the disease and these levels rise as the disease progresses. The researchers were screening for genes that are regulated by the circadian rhythm, and were intrigued to see the gene for this brain protein pop up.

“The gene for YKL-40 came up as highly regulated by clock genes,” says Erik Musiek, senior author. “That was really interesting because it is a well-known biomarker for Alzheimer’s.”

Chitinase-3-like protein 1 (CHI3L1), also known as YKL-40, is a secretedglycoprotein that is approximately 40kDa in size that in humans is encoded by the CHI3L1gene.[5][6][7] The name YKL-40 is derived from the three N-terminal amino acids present on the secreted form and its molecular mass. YKL-40 is expressed and secreted by various cell-types including macrophageschondrocytes, fibroblast-like synovial cells, vascular smooth muscle cells, and hepatic stellate cells. The biological function of YKL-40 is unclear. It is not known to have a specific receptor. Its pattern of expression is associated with pathogenic processes related to inflammation, extracellular tissue remodeling, fibrosis and solid carcinomas[8] and asthma.[9]

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